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Chapter 1 beginning comments (pages 1–3): Arnold R. Rich
Chapter 2 Ultrastructure of the Glomerulus and adjustments in advantageous constitution linked to elevated Permeability of the Glomerulus to Protein (pages 4–31): Robert L. Vernier
Chapter three Glomerular harm when it comes to “Pore measurement” (pages 32–50): J. Hardwicke and J. F. Soothill
Chapter four Electron Microscopy of Renal Glomerular Amyloidosis (pages 51–69): A. Bergstrand and H. Bucht
Chapter five scientific, Microscopic and Electron Microscopic facts within the Nephrotic Syndrome of Unknown starting place (pages 70–102): R. Habib, P. Michielsen, E. De Montera, N. Hinglais, P. Galle and J. Hamburger
Chapter 6 The effective constitution of the Glomerulus in Bright's disorder: A Clinico?Pathological examine (pages 103–155): Henry Z. Movat, Jan W. Steiner and Robert J. Slater
Chapter 7 Post?Streptococcal Glomerulonephritis (pages 156–193): David P. Earle and Robert B. Jennings
Chapter eight Focal Glomerulonephritis (pages 194–217): R. H. Heptinstall and A. M. Joekes
Chapter nine Lupus Nephritis (pages 218–244): Conrad L. Pirani, Victor E. Pollak, Robert C. Muehrcke and Robert M. Kark
Chapter 10 Percutaneous Renal Biopsy in Pyelonephritis (pages 245–261): Claus Brun and Flemming Raaschou
Chapter eleven Correlation among Renal Biopsy and different Diagnostic systems in Pyelonephritis (pages 262–280): M. S. R. Hutt and H. E. De Wardener
Chapter 12 the importance of Renal Biopsy for the prognosis of Pyelonephritis in Diabetic sufferers (pages 281–308): Age Chr. Thomsen
Chapter thirteen Quantitative Histochemistry of the Nephron (pages 309–329): Robert M. Kark, Hermann Mattenheimer, Sjoerd L. Bonting, Victor E. Pollak and Robert C. Muehrcke
Chapter 14 a few Observations at the effective constitution of Human Kidney Biopsies in Acute Anuria and Osmotic Diuresis (pages 330–378): O. Z. Dalgaard and ok. J. Pedersen

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And DE HEINZELIN DE BRANCORT, C. (1952). Arch. int. , 60, 506 KARK,R. , PIRANI, C. , POLLAK, V. , MUEHRCKE, R. , and BLAINEY, J. (1958). Ann. intern. , 49, 751. -P. (1957). franf. &t. clin. b i d , 2, 145. MALMENDIER, C. -P. (1955). J. Vrol. (Baltimore), 61,327. MONKE, J. V. and YUILE,C. L. (1940). J. exp. , 72, 149. PAPPENHEIMER, J. R. (1955). Klin. , 33, 362. PHELPS, R. A. and PUTNAM, F. W. (1960). In The Plasma Proteins, Vol. I , p. 159. New York and London: Academic Press. SCH~NENBERGER, V.

Hnrdwicke: That is the suggestion from the functional studies. If one is working on Pappenheimer’s diffusion theory, the longer you have the blood in the capillary the more chance there is for complete equilibration of partially filtered colloids across the filter. Thus thelonger the blood is in the capillary the inore protein is going to diffuse out into Bowman’s space. It is striking that in these patients, particularly in children, the capillariesare nearly always grossly dilated and blood-filled.

This reasoning depends on the relative composition of the proteins in the glomerular filtrate being the same as that in the urine; specific reabsorption in the tubules of any protein being studied would invalidate the above conclusions. However, on following the rise in albumin clearance after intravenous albumin infusion, it was found that the clearances of the globulins also rose iii a constant ratio to the albumin clearance. This is interpreted as showing that reabsorption is not selective between the different proteins, and therefore the urine has the same relative composition of individual protein fractions as the glomerular filtrate, though, of course, the total quantity excreted is less by the amount of any protein reabsorption occurring in the tubules (Hardwicke and Squire, 1955).

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